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AHR — FOS
Pathways - manually collected, often from reviews:
Text-mined interactions from Literome
Suh et al., Toxicol Appl Pharmacol 2002
(Lymphoma) :
Aryl hydrocarbon receptor dependent inhibition of
AP-1 activity by 2,3,7,8-tetrachlorodibenzo-p-dioxin in activated B cells ... Collectively, these results suggest that TCDD induced inhibition of IgM expression by B cells may be mediated, at least in part, through a down-regulation of
AP-1 activity in an
AhR dependent manner
Kizu et al., Arch Toxicol 2003
(Prostatic Neoplasms) :
These results suggest that
AhR can
stimulate AP-1 expression resulting in inhibition of the binding of AR to ARE in the transcription regulatory region of target genes such as PSA