◀ Back to IGF1

IGF1 — VIP

Text-mined interactions from Literome

Servoss et al., Endocrinology 2001 : Both VIP- and activity dependent neurotrophic factor stimulated growth were blocked by anti-IGF-I , whereas anti-activity dependent neurotrophic factor-14 had no detectable effect on IGF-I induced growth
Fernández et al., Neuroendocrinology 2003 : In previous studies we demonstrated that insulin-like growth factor I (IGF-I) induces pituitary vasoactive intestinal peptide (VIP) gene expression and secretion, and that IGF-I induced prolactin (PRL) release is mediated by VIP ... These results indicate that both IGF-I and VIP stimulate lactotrope proliferation, and that IGF-I induced lactotrope proliferation is partially mediated by VIP produced locally
Saitoh et al., Cell Tissue Res 2010 : In addition, no vasoactive intestinal peptide (VIP)-immunoreactive cells were found in KO mice, therefore IGF-I is essential for VIP expression
Lara et al., Endocrinology 1994 : The effects of recombinant human insulin-like growth factor I (IGF-I) on both vasoactive intestinal peptide (VIP) and PRL production and gene expression were studied using rat anterior pituitary cell cultures grown in serum-free defined medium ... In conclusion, these results indicate that IGF-I induces VIP gene expression, and its secretion and also increases PRL secretion ... The effect of IGF-I on PRL release is specifically mediated by VIP through a paracrine or autocrine mechanism