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FOS — PDCD4

Text-mined interactions from Literome

Lehmann et al., Cell Death Differ 2002 : This study demonstrates a role for AP-1 in the stage-specific steroid triggered programmed cell death of larval tissues during Drosophila metamorphosis
Zahradnícková et al., Cell Mol Life Sci 2003 (Cell Transformation, Neoplastic...) : c-Fos but not v-Fos protein induces programmed cell death of v-myb transformed monoblasts ... We show that while the v-Fos protein prolongs the G0G1 phase of the BM2 cell cycle, c-Fos leaves the cell cycle unaffected and, rather, induces programmed cell death
Jansen et al., Cancer Res 2005 (Carcinoma, Squamous Cell...) : These results extend to an in vivo model the observations that Pdcd4 inhibits both translation initiation and AP-1 activation while decreasing benign tumor development and malignant progression
Palamarchuk et al., Cancer Res 2005 : Using luciferase assay, we show that phosphorylation of Pdcd4 by Akt also causes a significant decrease of the ability of Pdcd4 to interfere with the transactivation of AP-1-responsive promoter by c-Jun
Talotta et al., Oncogene 2009 (Cell Transformation, Neoplastic) : We further show that, given the role of PDCD4 as negative regulator of AP-1, the miR-21 mediated downregulation of PDCD4 is essential for the maximal induction of AP-1 activity in response to RAS
Marti et al., Oncogene 1994 : A similar induction of AP-1 ( cFos/JunD ) was also observed in the involuting rat ventral prostate pointing to a possible role for AP-1 in programmed cell death
Feng et al., Oncogene 1998 (Atrophy) : Our data strongly suggest that c-Fos is required for programmed cell death of prostate epithelial cells