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INS — LIMK1

Text-mined interactions from Literome

Rosso et al., Mol Biol Cell 2004 : Transfection of wild-type LIMK1 accelerates axon formation and enhances the accumulation of Par3/Par6, insulin-like growth factor (IGF)1 receptors, and neural cell adhesion molecule ( NCAM ) at growth cones, while inhibiting the Golgi export of synaptophysin containing vesicles
Tang et al., Environ Toxicol 2007 : Similarly, insulin restored the decrease of LIMK-1 and cofilin phosphorylation induced by A 1254