Gene interactions and pathways from curated databases and text-mining

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CXCL10 — CXCL5

Pathways - manually collected, often from reviews:

Text-mined interactions from Literome

Schuh et al., J Immunol 2002 (Asthma...) : Interestingly, at day 3 after conidia, neutrophil recruitment and airway hyperresponsiveness in CXCR2-/- mice was mediated by inflammatory protein-10/CXCL10 and, to a lesser degree, MIG/CXCL9
Sharma et al., Molecular cancer 2003 (Carcinoma, Lewis Lung) : In vivo depletion of IP-10/CXCL10 , MIG/CXCL9 or IFNgamma significantly reduced the anti-tumor efficacy of SLC/CCL21
Kalwitz et al., Int J Biochem Cell Biol 2009 : Induction of differentially expressed chemokines CXCL1-3, CXCL5 , and CXCL6 as well as LIF and gp130 in MSC by CXCL7 was verified by real-time polymerase chain reaction
McKimmie et al., Biochem Biophys Res Commun 2010 : Bacterial associated molecules induced the preferential expression of CCL2, CXCL1 , CCL20 and CCL3 whilst a virus associated dsRNA analogue preferentially up-regulated CXCL10 and CCL5
Maity et al., Cytokine 2011 : For example, CXCL8 expression was markedly enhanced ( p < 0.05 ) whereas CXCL5 expression was significantly repressed ( p < 0.05 ) in cells exposed to HS coincident with TNFa stimulation
Geluk et al., J Immunol 2011 (Leprosy) : Most CD8 ( + ) T cells produced IFN-?, but distinct IFN-? ( + ) /TNF-a ( + ) populations were detected simultaneously with significant secretion of CXCL10/IFN-? induced protein 10, CXCL9/MIG , and VEGF