Gene interactions and pathways from curated databases and text-mining

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ADCY5 — PI3

Text-mined interactions from Literome

Niwa et al., Life Sci 2000 : Both prostaglandin E2 ( PGE2 ) and wortmannin, an inhibitor of phosphatidyl inositol 3-kinase (PI-3K), inhibited neutrophil aggregation, whereas dbcAMP, a cell permeable analog of cyclic AMP, did not, confirming that PGE2 causes neutrophil aggregation probably through PI-3K inhibition rather than activation of adenylate cyclase
Hardy et al., Blood 2004 (Calcium Signaling) : This potentiation is mediated by 2 mechanisms : inhibition of adenylate cyclase and activation of phosphatidylinositol 3 (PI 3)-kinase
Carini et al., Gastroenterology 2004 (Reperfusion Injury) : In the cells treated with CGS21680, PI3K activation was prevented either by inhibiting adenylate cyclase and PKA with, respectively, 2,5-dideoxyadenosine and H89 or by blocking Galphai-protein and Src tyrosine kinase with, respectively, pertussis toxin and PP2
Hildebrand et al., Cell communication and signaling : CCS 2012 : PMT inhibits the production of IL-12p40 by Gai mediated inhibition of adenylate cyclase and cAMP accumulation and by Gß? mediated activation of PI3kinase and JNK activation