◀ Back to CCND1

BAX — CCND1

Text-mined interactions from Literome

Liu et al., Int J Cancer 2008 (Body Weight...) : Proliferating cell nuclear antigen ( PCNA ), cyclin D1 and Bcl-2 expression decreased, and Bax expression and nuclear fragmentation increased with increasing dietary beta-ionone
Yang et al., J Nutr 2008 (Adenomatous Polyposis Coli...) : Feeding WD to Apc ( 1638N/+ ) mice not only enhanced cyclin D1 expression in colonic epithelium compared with AIN-76A treatment as previously reported but also significantly increased the expression of the antiapoptotic protein B-cell lymphoma 2 (Bcl-2) concomitantly with a decrease in the proapoptotic Bcl2 associated X protein and the number of apoptotic epithelial cells
Festuccia et al., Endocr Relat Cancer 2009 (Disease Models, Animal...) : In vivo, azacitidine ( 0.8 mg/kg i.p. ) reduced tumor proliferation and induced apoptosis in both xenografts upmodulating the expression of p16INKA, Bax , Bak, p21/WAF1, and p27/KIP1, and inhibiting the activation of Akt activity and the expression of cyclin D1 , Bcl-2, and Bcl-XL
Kim et al., Arch Pharm Res 2009 (Colonic Neoplasms) : NF-kappaB target gene expression of apoptotic cell death proteins ( Bax , caspase-3, caspase-9 ) was significantly enhanced, but the expression of anti-apoptotic genes and cell proliferation marker genes ( Bcl-2, inhibitor of apoptosis protein ( IAP-1 ) and X chromosome IAP (XIAP), Cox-2, c-Fos, c-Jun and cyclin D1 ) was significantly inhibited by the combined treatment compared to Rg3 or docetaxel alone
Ganapathy et al., PloS one 2010 (Neoplasm Metastasis...) : In xenografted tumors, resveratrol upregulated the expressions of TRAIL-R1/DR4, TRAIL-R2/DR5, Bax and p27 ( /KIP1 ), and inhibited the expression of Bcl-2 and cyclin D1
Bergeron et al., Curr Opin Neurobiol 1998 (Nervous System Diseases) : When neurons are deprived of trophic factors, a sequence of events is initiated, which includes a reduction in macromolecule synthesis, elevation of c-Jun and cyclin D1 , and activation of BAX