Gene interactions and pathways from curated databases and text-mining

◀ Back to ICAM1

ICAM1 — IFNB1

Text-mined interactions from Literome

Farrokh-Siar et al., Curr Eye Res 2001 : IFN-beta and IL-beta1 only up-regulated the expression of ICAM-1
Fujisawa et al., J Dermatol 1992 (Carcinoma, Squamous Cell...) : It has been previously reported that IFN-beta suppresses IFN-gamma induced ICAM-I expression in A431 cells, a human squamous cell carcinoma cell line
Reddy et al., J Natl Cancer Inst 1991 (Glioblastoma) : Of the antigens tested, ICAM-1 and HLA class I antigens were the most sensitive to enhanced expression induced by IFN-beta and IFN-gamma when used alone or in combination
Kobayashi et al., Eur Respir J 2008 (Inflammation) : IFN-beta significantly enhanced the expression of vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 on HUVECs in the presence of TNF-alpha
Horikoshi et al., FEBS Lett 1995 : Up-regulation of ICAM-1 expression on human dermal fibroblasts by IFN-beta in the presence of TNF-alpha ... Interferon-beta (IFN-beta) at concentrations of 10, 100, and 1000 IU/ml in the presence of tumor necrosis factor-alpha (TNF-alpha) significantly increased the ICAM-1 expression of fibroblasts in a dose dependent manner
Satoh et al., Neurology 1995 : We investigated the modulatory effects of human interferon beta (IFN-beta) and gamma ( IFN-gamma ) on expression of class I and II major histocompatibility complex ( MHC ) antigens and intercellular adhesion molecule-1 ( ICAM-1 ) in fetal human astrocytes in culture using flow cytometry and immunocytochemistry ... However, IFN-beta did not significantly induce expression of class II MHC antigen or ICAM-1 ... Furthermore, IFN-beta significantly reduced IFN-gamma induced expression of class II MHC antigen but not of class I MHC antigen or of ICAM-1 ... The differential effects of IFN-beta and IFN-gamma on expression of class I and II MHC antigens and ICAM-1 in fetal human astrocytes suggest that interferons serve as modulators of astrocyte function at sites of inflammation in the human CNS
Miller et al., J Neuroimmunol 1996 (Autoimmune Diseases...) : As vascular endothelial cells are active participants in inflammatory and demyelinating processes, we decided to examine the effects of IFN-beta on the expression of major histocompatibility complex ( MHC ) gene products and intercellular adhesion molecule (ICAM)-1 on human vascular endothelial cells ( ECs ) ... Basal expression of ICAM-1 molecules was enhanced by TNF alpha and to a lesser extent by IFN-beta , but was not affected by IFN-gamma
Jiang et al., Neurochem Int 1997 : Since adhesion molecules ( AM ) induced on endothelial cells ( EC ) play an important role in T-cell migration into the CNS, the objective of this study was to examine the effect of IFN beta-1b on the expression of the AM, ICAM-1 , V-CAM and E-selection induced on EC by IFN-gamma, TNF-alpha, or IL-1 beta ... Since adhesion molecules ( AM ) induced on endothelial cells ( EC ) play an important role in T-cell migration into the CNS, the objective of this study was to examine the effect of IFN beta-1b on the expression of the AM, ICAM-1 , V-CAM and E-selection induced on EC by IFN-gamma, TNF-alpha, or IL-1 beta