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JUN — PCNA

Text-mined interactions from Literome

Clark et al., Mol Cell Biol 2000 : Despite this, v-Jun does not stimulate D-cyclin-cdk activity but does induce a marked deregulation of cyclin E-cdk2 ... In particular, hormonal activation of a conditional v-Jun-estrogen receptor fusion protein in quiescent, growth factor deprived cells stimulates cyclin E-cdk2 activity and triggers Rb phosphorylation and DNA synthesis
Besirli et al., J Biol Chem 2003 (MAP Kinase Signaling System) : Pharmacological inhibitors of cyclin dependent kinase (CDK) activity completely prevented c-Jun phosphorylation after DNA damage
Berglund et al., J Neurosci Res 2004 (Neuroblastoma) : Possible involvement of cyclin dependent kinases (CDKs) in c-Jun phosphorylation at Ser63 was excluded by pretreating UV-irradiated SH-SY5Y cells with the CDK1/2/5 inhibitor roscovitine
Lee et al., J Biomed Sci 2008 (Colitis...) : In addition, consumption of UDN glycoprotein attenuated the activities of proliferating cell nuclear antigen ( PCNA ), inducible nitric oxide synthase (iNOS), and cyclooxygenase-2 (COX-2), and inhibited the DNA binding activities of nuclear factor-kappa B (NF-kappaB) and activator protein-1 (AP-1) in the mice colonic tissue
Lorca et al., EMBO J 1991 : Degradation of the proto-oncogene product p39mos is not necessary for cyclin proteolysis and exit from meiotic metaphase : requirement for a Ca ( 2+ ) -calmodulin dependent event
Ni et al., J Biol Chem 2010 (Hyperplasia) : Adenovirus forced c-Jun expression stimulated SMC proliferation, proliferating cell nuclear antigen , and MMP-2 expression