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MYLIP — NOS2
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Suárez et al., Circ Res 2007
:
The miRNA expression profile of EC revealed 25 highly expressed miRNAs in human EC and using miRNA mimicry,
miR-222/221 regulates endothelial
nitric oxide synthase protein levels after Dicer silencing
Dai et al., Blood 2008
:
miR-223 , which is markedly enhanced by estrogen,
regulates LPS induced IFNgamma, but not
iNOS or nitric oxide in splenic lymphocytes
Wang et al., J Biol Chem 2009
:
Transfection of
miR-155 attenuated the expression of Suppressor of Cytokine Signal (SOCS)-1 and
enhanced the expression of
iNOS ... Our results suggest that MKP-1 may negatively
regulate iNOS expression by controlling the expression of
miR-155 and consequently the STAT pathway via SOCS-1
Abouheif et al., Rheumatology (Oxford) 2010
(Arthritis, Experimental) :
Silencing of
miR-34a significantly
prevented IL-1ß induced down-regulation of Col2a1, as well as IL-1ß induced up-regulation of
iNOS
Perske et al., Am J Pathol 2010
(Carcinoma, Renal Cell...) :
Loss of inducible
nitric oxide synthase expression in the mouse renal cell carcinoma cell line RENCA is
mediated by microRNA
miR-146a
Yamaguchi et al., Atherosclerosis 2011
:
In ES-ECs,
miR-145 overexpression did not induce the synthesis of smooth muscle related proteins nor
suppress the expression of endothelial
nitric oxide synthase
Boon et al., Thromb Haemost 2012
(Atherosclerosis) :
The down-regulation of
miR-92a by shear stress
enhances the expression of the endothelial
nitric oxide synthase , whereas the up-regulation of miR-19a contributes to the shear stress induced inhibition of cell proliferation
Shi et al., Glia 2013
(Inflammation...) :
The overexpression of
miR-195 contributed to lipopolysaccharide induced expression of proinflammatory cytokines IL-1ß, TNF-a, and
iNOS in cultured microglia