Gene interactions and pathways from curated databases and text-mining

◀ Back to MYLIP

MYLIP — NOS2

Text-mined interactions from Literome

Suárez et al., Circ Res 2007 : The miRNA expression profile of EC revealed 25 highly expressed miRNAs in human EC and using miRNA mimicry, miR-222/221 regulates endothelial nitric oxide synthase protein levels after Dicer silencing
Dai et al., Blood 2008 : miR-223 , which is markedly enhanced by estrogen, regulates LPS induced IFNgamma, but not iNOS or nitric oxide in splenic lymphocytes
Wang et al., J Biol Chem 2009 : Transfection of miR-155 attenuated the expression of Suppressor of Cytokine Signal (SOCS)-1 and enhanced the expression of iNOS ... Our results suggest that MKP-1 may negatively regulate iNOS expression by controlling the expression of miR-155 and consequently the STAT pathway via SOCS-1
Abouheif et al., Rheumatology (Oxford) 2010 (Arthritis, Experimental) : Silencing of miR-34a significantly prevented IL-1ß induced down-regulation of Col2a1, as well as IL-1ß induced up-regulation of iNOS
Perske et al., Am J Pathol 2010 (Carcinoma, Renal Cell...) : Loss of inducible nitric oxide synthase expression in the mouse renal cell carcinoma cell line RENCA is mediated by microRNA miR-146a
Yamaguchi et al., Atherosclerosis 2011 : In ES-ECs, miR-145 overexpression did not induce the synthesis of smooth muscle related proteins nor suppress the expression of endothelial nitric oxide synthase
Boon et al., Thromb Haemost 2012 (Atherosclerosis) : The down-regulation of miR-92a by shear stress enhances the expression of the endothelial nitric oxide synthase , whereas the up-regulation of miR-19a contributes to the shear stress induced inhibition of cell proliferation
Shi et al., Glia 2013 (Inflammation...) : The overexpression of miR-195 contributed to lipopolysaccharide induced expression of proinflammatory cytokines IL-1ß, TNF-a, and iNOS in cultured microglia