Gene interactions and pathways from curated databases and text-mining

◀ Back to BCL6

BCL6 — PDCD2

Text-mined interactions from Literome

Pinto et al., J Comput Aided Mol Des 2004 : Overexpression of Bcl-2 and Bcl-xL proteins, both inhibitors of apoptosis or programmed cell death , is related to the generation and development of several types of cancer as well as to an elevated resistance to chemotherapeutic treatments
Peterson et al., Hybridoma (Larchmt) 2005 : Annexin staining in hybridoma 7.16.4 confirmed that apoptosis was the primary means of cell death in this cell line, and expression of Bcl-2-delta and Bcl-xl-delta inhibited programmed cell death
Thuduppathy et al., Protein Sci 2006 : Regulation of programmed cell death by Bcl-xL is dependent on both its solution and integral membrane conformations
Schäbitz et al., J Cereb Blood Flow Metab 2008 (Brain Infarction...) : In primary cortical neurons and human neuroblastoma cells, GM-CSF counteracts programmed cell death and induces BCL-2 and BCL-Xl expression in a dose- and time dependent manner
Baron et al., Proc Natl Acad Sci U S A 2007 (Lymphoma, B-Cell...) : Repression of the PDCD2 gene by BCL6 and the implications for the pathogenesis of human B and T cell lymphomas ... These data confirm PDCD2 as a target of BCL6 and support the concept that repression of PDCD2 by BCL6 is likely important in the pathogenesis of certain human lymphomas
Li et al., J Biol Chem 1997 : Bcl-xL , an antiapoptotic member of the Bcl-2 family, inhibits programmed cell death in a broad variety of cell types