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EPHB2 — TCF24

Text-mined interactions from Literome

Yoshizumi et al., Jpn J Pharmacol 2000 : The ERK1/2 activation was followed by an increase in transcription factor activator protein-1 DNA binding activity
Wang et al., J Biol Chem 2002 : Superoxide mediates shock wave induction of ERK dependent osteogenic transcription factor ( CBFA1 ) and mesenchymal cell differentiation toward osteoprogenitors
Ying et al., J Neurosci 2002 : BDNF-LTP was further coupled to ERK dependent phosphorylation of the transcription factor cAMP response element binding protein
Wang et al., J Biol Chem 2004 (Neovascularization, Pathologic) : Ras induction of superoxide activates ERK dependent angiogenic transcription factor HIF-1alpha and VEGF-A expression in shock wave stimulated osteoblasts
Mao et al., Eur J Neurosci 2004 : In a further attempt to evaluate the role of active ERK1/2 in activating a downstream transcription factor cAMP response element binding protein ( CREB ), NMDA, AMPA, and kainate were found to increase CREB phosphorylation
Kim et al., Arch Pharm Res 2004 : Several reports indicate that the activation of extracellular signal regulated kinase ( ERK ) induces microphthalmia associated transcription factor ( MITF ) degradation
Ingram et al., Kidney Int 2004 (MAP Kinase Signaling System) : Erk induces increases in activator protein-1 (AP-1) transcription factor activity which may augment mesangial cell proliferation and ECM protein production
Song et al., J Biol Chem 2005 : IFN-gamma induced ERK1/2 phosphorylation and subsequently the transcription factor early gene response 1, which in turn up-regulated p35 expression and increased cyclin dependent kinase 5 (Cdk5) activity
Haberny et al., Neuroscience 2005 : These results point to specific neuroadaptations in the NAc of FR rats whereby D-1 DA receptor stimulation leads to increased NMDA NR1 subunit phosphorylation and consequent increases in NMDA receptor dependent CaMK II and ERK1/2 signaling, and increased NMDA receptor/ERK1/2 dependent phosphorylation of the nuclear transcription factor , CREB
Molina et al., J Biol Chem 2005 : Furthermore, D-site peptides, which are able to potently inhibit ERK2 mediated phosphorylation of the Elk-1 transcription factor ( IC50= 3 microm ), are relatively poor inhibitors of ERK2 mediated phosphorylation of MITF, exhibiting > 15-fold selectivity for inhibition of Elk-1 versus MITF
Maclachlan et al., Int J Radiat Biol 2005 (Radiation Injuries) : In vitro exposure to FGF-20 increased expression of the Nrf2 transcription factor and oxygen radical scavenging enzymes such as MnSOD, activated signal transduction pathways ( ERK and Akt ) and resulted in increased survival of irradiated cells in vitro
Gelain et al., Cell Signal 2006 : In this work, we report that, in Sertoli cells, retinol ( 7 microM ) induces the Src dependent activation of ERK1/2 MAPK and the ERK1/2 mediated phosphorylation of the transcription factor CREB
Park et al., Neurosci Lett 2006 (Neuroblastoma) : The mitogen activated protein kinase/extracellular signal regulated kinase ( MAPK/ERK ) activates a transcription factor CREB
Zhang et al., J Immunol 2006 : Blocking the activation of ERK prevented histone phosphorylation and transcription factor binding to the IL-10 promoter
Cortez et al., Am J Physiol Heart Circ Physiol 2007 : IL-17 induced p38 MAPK and ERK1/2 activation , and inhibition by SB-203580 and PD-98059 blunted IL-17 mediated transcription factor activation and MMP-1 expression
Kilanczyk et al., Biochem Biophys Res Commun 2009 : We also demonstrate that CacyBP/SIP-ERK1/2 interaction inhibits phosphorylation of the Elk-1 transcription factor in vitro and in the nuclear fraction of NB2a cells
Xu et al., Biochem Biophys Res Commun 2009 : We now show in both HEK cells and human microglial cells that the induction of Cyr61 protein expression by the human FP receptor utilizes a novel mechanism involving the activation of Ras and Raf followed by a MEK/ERK independent activation of Tcf signaling
Biyashev et al., J Biol Chem 2010 (MAP Kinase Signaling System) : We demonstrate that Erk5 phosphorylation allows activation of PPARgamma transcription factor by displacement of SMRT co-repressor
Cook et al., Am J Surg 2010 (Thyroid Neoplasms) : Xanthohumol inhibits the neuroendocrine transcription factor achaete-scute complex-like 1, suppresses proliferation, and induces phosphorylated ERK1/2 in medullary thyroid cancer
Bae et al., Nephrol Dial Transplant 2011 : HHE mediated accumulation of ROS may induce redox-sensitive transcription factor , NF-?B, through activation of ERK and JNK, resulting in cellular apoptosis in HK-2 cells
Wu et al., Mol Cancer Res 2011 (Breast Neoplasms...) : LPA stimulated p21 via LPA ( 1 ) and LPA ( 2 ) receptors and Erk dependent activation of the CCAAT/enhancer binding protein beta transcription factor independent of p53
Yen et al., J Biol Chem 2011 (MAP Kinase Signaling System) : PKA, PI3K, and ERK inhibitors abolished PGE2- and cAMP induced c-Fos and MMP-9 up-regulation, and ERK activation was required for the binding of activator protein 1 (AP-1) transcription factor to the MMP-9 promoter
Zhang et al., Mol Cell Biochem 2013 (MAP Kinase Signaling System) : ROS production leads to activation of ERK1/2 and then activation of the NF-?B transcription factor
Janknecht et al., Mol Cell Biol 1996 : Analysis of the ERK stimulated ETS transcription factor ER81
Yoshizumi et al., Br J Pharmacol 1998 : 4. Gel-mobility shift analysis revealed that the ERK1/2 activation was followed by an increase in transcription factor activator protein-1 DNA binding activity in HCASMCs