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IRF6 — TFPI

Text-mined interactions from Literome

Sautebin et al., Inflamm Res 1999 : AA induced a significant production of 6-ketoPGF1alpha and PGE2, whereas LPS caused a concentration- and time dependent increase of 8-epi-PGF2alpha , 6-keto-PGF1alpha and PGE2 ... DXM ( 2 microM ) as well as CXM ( 1 microM ) significantly decreased ( p < 0.001 ; n = 4 ) the LPS stimulated production of 8-epi-PGF2alpha ( by 86 % and 82 %, respectively ), 6-ketoPGF1alpha ( by 78 % and 74 %, respectively ) and PGE2 ( by 83 % and 78 %, respectively )
Aharon et al., Thromb Haemost 2004 : LPS and TNFalpha increased the high constitutive TF in STB, whereas LPS and IL-1alpha further reduced TFPI levels
Ollivier et al., Thromb Haemost 2005 (Inflammation) : Rapamycin slightly inhibits TFPI induction by LPS in monocytic cells
Birnbaum et al., Prostaglandins Other Lipid Mediat 2007 : LPS increased 15-epi-LXA4 and 6-keto-PGF ( 1alpha ) levels ... ASA ( 50 mg/kg and 200 mg/kg, but not 10 mg/kg ) augmented the LPS effect on 15-epi-LXA4 but attenuated the effect on 6-keto-PGF ( 1alpha )
Hsu et al., Lab Invest 2010 (Colitis) : LPS selectively induces EPI and AR but not other EGFR ligands ... LPS induced early EPI mRNA expression between 30 min and 24 h
Johnson et al., Blood 1996 (Inflammation) : Coagulation induced cytokine production and the synergistic production of IL-8 by coagulation and LPS could be attenuated by hirudin or tissue factor pathway inhibitor (TFPI)